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Performance comparison

CJC-1295 vs Follistatin 344

An educational, source-based comparison of CJC-1295 and Follistatin 344 — how each peptide works, what it's researched for, and what to know before going deeper.

Growth Hormone Axis
CJC-1295

GHRH analog often paired with Ipamorelin for pulsatile support.

Mechanism

A synthetic analog of growth hormone-releasing hormone (GHRH). The non-DAC version has a short half-life that preserves pulsatile GH release, while DAC variants extend the half-life via albumin binding. Research focuses on stimulating endogenous GH and IGF-1 production via the pituitary.

Research areas
  • Endogenous GH pulse amplitude
  • IGF-1 elevation
  • Body composition in adult GH-deficient research
  • Sleep architecture (slow-wave sleep)
Considerations
  • GH-axis manipulation may affect glucose tolerance.
  • Banned in competitive sport (WADA).
  • Requires physician oversight in any therapeutic context.
Full CJC-1295 profile →
Muscle · Myostatin Inhibition
Follistatin 344

Myostatin-binding protein fragment researched for muscle growth.

Mechanism

A naturally occurring glycoprotein that binds and neutralizes myostatin and activin, releasing the natural brake on skeletal muscle growth in preclinical models.

Research areas
  • Muscular dystrophy models
  • Skeletal muscle hypertrophy
  • Age-related sarcopenia (preclinical)
Considerations
  • Off-target effects on activin signaling (reproductive, cardiac) are a concern.
  • Not FDA-approved; banned by WADA.
Full Follistatin 344 profile →

CJC-1295 vs Follistatin 344 — Key differences

  • Class: CJC-1295 is classified as Growth Hormone Axis, while Follistatin 344 is Muscle · Myostatin Inhibition.
  • Primary research focus: CJC-1295endogenous gh pulse amplitude; Follistatin 344muscular dystrophy models.
  • Tag: Growth hormone vs Muscle.

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